carbon monoxide detector 50 ppm image
Answer
If it truly is only 2 ppm then you are within the limits of OSHA which has established a 50 PPM limit for the workplace.... But be careful of the testing device you are using. Some devices use a different unit of measure. The safest concentration is zero.
Some detection devices only measure concentrations of 1,000 parts per million and higher, significantly above safe levels. Testing equipment should be capable of sensing levels as low as one part per million. For example, Underwriters Laboratories' standard for residential carbon monoxide detectors requires detectors to alarm before 90 minutes of exposure to 100 parts per million of carbon monoxide.
If initial readings don't reveal sufficient concentrations of carbon monoxide to set off the alarm, digital measurement testing equipment that produces a printed 24-hour record can be used to help identify the source.
If it truly is only 2 ppm then you are within the limits of OSHA which has established a 50 PPM limit for the workplace.... But be careful of the testing device you are using. Some devices use a different unit of measure. The safest concentration is zero.
Some detection devices only measure concentrations of 1,000 parts per million and higher, significantly above safe levels. Testing equipment should be capable of sensing levels as low as one part per million. For example, Underwriters Laboratories' standard for residential carbon monoxide detectors requires detectors to alarm before 90 minutes of exposure to 100 parts per million of carbon monoxide.
If initial readings don't reveal sufficient concentrations of carbon monoxide to set off the alarm, digital measurement testing equipment that produces a printed 24-hour record can be used to help identify the source.
Carbon Monoxide and homeostasis?
Q. If the bond formed between carbon monoxide and hemoglobin is over 200 time stronger than the bond formed between oxygen and hemoglobin, how does exposure to carbon monoxide affect homeostasis?
Answer
Hemoglobin transports oxygen and carbon dioxide back and forth. Depending on the concentration in each environment, CO2 or O2 will break away from hemoglobin and diffuse into the cells or into lungs.
But if a certain molecule is bound to hemoglobin very tightly, they will not break away easily to diffuse into cells or into the lung. And Carbon monoxide is a molecule that will bind hemoglobin and leave no space for the much needed CO2 and O2 molecules to bind to hemoglobin.
Thus all hemoglobin when bound by Carbon monoxide will not leave any space for CO2 or O2 to bind. Thus there will be no cellular respiration occuring. Thus a person will die.
Carbon monoxide is a significantly toxic gas and has no odor or color. It is the most common type of fatal poisoning in many countries. Exposures can lead to significant toxicity of the central nervous system and heart. Following poisoning, long-term sequelae often occurs. Carbon monoxide can also have severe effects on the foetus of a pregnant woman. Symptoms of mild poisoning include headaches and dizziness at concentrations less than 100 ppm. Concentrations as low as 667 ppm can cause up to 50% of the body's haemoglobin to be converted to carboxy-haemoglobin (HbCO). Carboxy-haemoglobin is quite stable but this change is reversible. Carboxy-haemoglobin is ineffective for delivering oxygen, resulting in some body parts not receiving oxygen needed. As a result, exposures of this level can be life-threatening. In the United States, OSHA limits long-term workplace exposure levels to 50 ppm.
The mechanisms by which carbon monoxide produces toxic effects are not yet fully understood, but haemoglobin, myoglobin, and mitochondrial cytochrome oxidase are thought to be compromised. Treatment largely consists of administering 100% oxygen or hyperbaric oxygen therapy, although the optimum treatment remains controversial. Domestic carbon monoxide poisoning can be prevented by the use of household carbon monoxide detectors.
Hemoglobin transports oxygen and carbon dioxide back and forth. Depending on the concentration in each environment, CO2 or O2 will break away from hemoglobin and diffuse into the cells or into lungs.
But if a certain molecule is bound to hemoglobin very tightly, they will not break away easily to diffuse into cells or into the lung. And Carbon monoxide is a molecule that will bind hemoglobin and leave no space for the much needed CO2 and O2 molecules to bind to hemoglobin.
Thus all hemoglobin when bound by Carbon monoxide will not leave any space for CO2 or O2 to bind. Thus there will be no cellular respiration occuring. Thus a person will die.
Carbon monoxide is a significantly toxic gas and has no odor or color. It is the most common type of fatal poisoning in many countries. Exposures can lead to significant toxicity of the central nervous system and heart. Following poisoning, long-term sequelae often occurs. Carbon monoxide can also have severe effects on the foetus of a pregnant woman. Symptoms of mild poisoning include headaches and dizziness at concentrations less than 100 ppm. Concentrations as low as 667 ppm can cause up to 50% of the body's haemoglobin to be converted to carboxy-haemoglobin (HbCO). Carboxy-haemoglobin is quite stable but this change is reversible. Carboxy-haemoglobin is ineffective for delivering oxygen, resulting in some body parts not receiving oxygen needed. As a result, exposures of this level can be life-threatening. In the United States, OSHA limits long-term workplace exposure levels to 50 ppm.
The mechanisms by which carbon monoxide produces toxic effects are not yet fully understood, but haemoglobin, myoglobin, and mitochondrial cytochrome oxidase are thought to be compromised. Treatment largely consists of administering 100% oxygen or hyperbaric oxygen therapy, although the optimum treatment remains controversial. Domestic carbon monoxide poisoning can be prevented by the use of household carbon monoxide detectors.
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