osha carbon monoxide detector image
still_hvnt
My Mom and her coworkers have all suffered from carbon monoxide poisoning in the building where they work (this is in Texas). Four of them have nearly lost their lives now. The agency is covering it up and denying anything is wrong with the building, and are not fixing the problem. However it is obvious something is wrong - we have doctors reports, and a coworker's witness to an inspection from the Fire Marshall (in which he found toxic levels of carbon monoxide but it was not reported), as well as a coworker's personal Carbon Monoxide detector finding toxic traces of carbon monoxide in the building.... the list goes on.
This is a small town, the Fire marshall just so happens to be cousins with one of the managers at the agency who is covering it up. And apparently OSHA and the Health Department do not want to help either. We cannot get attorneys to call us back either.
Is there anything else we can we do to get help?
Answer
The fire department keeps logs of all calls. See if you can get a copy. Also, see if the police have any info on it. They must have been called if the fire dept. was. OSHA doesn't want to be involved? Have you gotten a lawyer yet? Co2 poisoning is so dangerous your mother and her coworkers were lucky to be alive. It is odorless and so easy to just close your eyes and die. Back to getting a lawyer. Get one that isn't in your town, they will know what to do. He can even all the documents. He can even go public, which is what the place won't want. OSHA should be involved. They almost died! Tell mom, get a lawyer. If they get away with this accident and deny it, they set a precedent for the next company to do the same. Do not back down. Best of luck to your mom and her coworkers.
The fire department keeps logs of all calls. See if you can get a copy. Also, see if the police have any info on it. They must have been called if the fire dept. was. OSHA doesn't want to be involved? Have you gotten a lawyer yet? Co2 poisoning is so dangerous your mother and her coworkers were lucky to be alive. It is odorless and so easy to just close your eyes and die. Back to getting a lawyer. Get one that isn't in your town, they will know what to do. He can even all the documents. He can even go public, which is what the place won't want. OSHA should be involved. They almost died! Tell mom, get a lawyer. If they get away with this accident and deny it, they set a precedent for the next company to do the same. Do not back down. Best of luck to your mom and her coworkers.
Carbon Monoxide and homeostasis?
Justafreak
If the bond formed between carbon monoxide and hemoglobin is over 200 time stronger than the bond formed between oxygen and hemoglobin, how does exposure to carbon monoxide affect homeostasis?
Answer
Hemoglobin transports oxygen and carbon dioxide back and forth. Depending on the concentration in each environment, CO2 or O2 will break away from hemoglobin and diffuse into the cells or into lungs.
But if a certain molecule is bound to hemoglobin very tightly, they will not break away easily to diffuse into cells or into the lung. And Carbon monoxide is a molecule that will bind hemoglobin and leave no space for the much needed CO2 and O2 molecules to bind to hemoglobin.
Thus all hemoglobin when bound by Carbon monoxide will not leave any space for CO2 or O2 to bind. Thus there will be no cellular respiration occuring. Thus a person will die.
Carbon monoxide is a significantly toxic gas and has no odor or color. It is the most common type of fatal poisoning in many countries. Exposures can lead to significant toxicity of the central nervous system and heart. Following poisoning, long-term sequelae often occurs. Carbon monoxide can also have severe effects on the foetus of a pregnant woman. Symptoms of mild poisoning include headaches and dizziness at concentrations less than 100 ppm. Concentrations as low as 667 ppm can cause up to 50% of the body's haemoglobin to be converted to carboxy-haemoglobin (HbCO). Carboxy-haemoglobin is quite stable but this change is reversible. Carboxy-haemoglobin is ineffective for delivering oxygen, resulting in some body parts not receiving oxygen needed. As a result, exposures of this level can be life-threatening. In the United States, OSHA limits long-term workplace exposure levels to 50 ppm.
The mechanisms by which carbon monoxide produces toxic effects are not yet fully understood, but haemoglobin, myoglobin, and mitochondrial cytochrome oxidase are thought to be compromised. Treatment largely consists of administering 100% oxygen or hyperbaric oxygen therapy, although the optimum treatment remains controversial. Domestic carbon monoxide poisoning can be prevented by the use of household carbon monoxide detectors.
Hemoglobin transports oxygen and carbon dioxide back and forth. Depending on the concentration in each environment, CO2 or O2 will break away from hemoglobin and diffuse into the cells or into lungs.
But if a certain molecule is bound to hemoglobin very tightly, they will not break away easily to diffuse into cells or into the lung. And Carbon monoxide is a molecule that will bind hemoglobin and leave no space for the much needed CO2 and O2 molecules to bind to hemoglobin.
Thus all hemoglobin when bound by Carbon monoxide will not leave any space for CO2 or O2 to bind. Thus there will be no cellular respiration occuring. Thus a person will die.
Carbon monoxide is a significantly toxic gas and has no odor or color. It is the most common type of fatal poisoning in many countries. Exposures can lead to significant toxicity of the central nervous system and heart. Following poisoning, long-term sequelae often occurs. Carbon monoxide can also have severe effects on the foetus of a pregnant woman. Symptoms of mild poisoning include headaches and dizziness at concentrations less than 100 ppm. Concentrations as low as 667 ppm can cause up to 50% of the body's haemoglobin to be converted to carboxy-haemoglobin (HbCO). Carboxy-haemoglobin is quite stable but this change is reversible. Carboxy-haemoglobin is ineffective for delivering oxygen, resulting in some body parts not receiving oxygen needed. As a result, exposures of this level can be life-threatening. In the United States, OSHA limits long-term workplace exposure levels to 50 ppm.
The mechanisms by which carbon monoxide produces toxic effects are not yet fully understood, but haemoglobin, myoglobin, and mitochondrial cytochrome oxidase are thought to be compromised. Treatment largely consists of administering 100% oxygen or hyperbaric oxygen therapy, although the optimum treatment remains controversial. Domestic carbon monoxide poisoning can be prevented by the use of household carbon monoxide detectors.
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